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Extrahepatic manifestations of chronic viral hepatitis

By systems such as hepatitis B patients developed Gemei Barre syndrome (1976, Niermciget), Farivat's other 1 case of chronic hepatitis with peripheral plastic! Du tomb plastic neuropathy exists in the blood serum containing IgM, IgG, H3sng, Dane particles condensation protein complexes. Furthermore, it was that when the optic nerve disorders and regulation of acute hepatitis with the reduced membrane binding protein 2 in chronic hepatitis C virus infection (1) with or without the ball cold eggs from the hyperlipidemia of cryoglobulinemia MPGN MPGN: EMC performance of multi-system symptoms, more often in women, characterized by joint pain, vasculitis, purpura, neuropathy, and glomerulonephritis. Xinjiang after 50 kidney patients is poor,[link widoczny dla zalogowanych], mainly glomerular damage is MPGN, based on geographical distribution, HCV infection in EMC mutate larger. Interferon therapy, serum HCVRNA cryoglobulinemia disappeared with the disappearance of clinical manifestations of EMC (joint pain, purpura, glomerulonephritis) were significantly related. Of non-cryoglobulinemia MPGN: patients often appear nephrotic syndrome, proteinuria and mild to moderate renal function is not base. Many patients with HCV infection GN i-type cryoglobulinemia, but 30 ~ 4O does not appear cryoglobulinemia, 70 to 75 with mild serum transaminase increased. Most patients, including examination of liver showed moderate degree of chronic hepatitis, may be associated with or without cirrhosis. Other relevant features include low complement experimental hyperlipidemia (CH5, Cs, and CA, a cycle of rheumatoid factor and ‰ higher circulating autoantibody test (anti-edge,[link widoczny dla zalogowanych], anti-smooth muscle, anti-neutrophil plasma) was generally low degree of positive or seep negative. glomerular renal plexus live drawing show cell structure to increase the number and lobular thread, but also shows mesangial proliferation and sclerosis. Most of the patients immunofluorescence showed IgM, Ig6 and c | deposition in the mesangial and capillary wall, electron microscopy characteristic MPGN endothelial deposited, its pathogenesis is unknown, and may include: ① chronic HCV infection leads to autoantibody formation, these autoantibodies and the inherent renal antigens lead to kidney physical change; @ chronic liver disease itself is a direct result of MPGN the occurrence of chronic liver disease associated with decreased reticuloendothelial cell function and (or) increase the immune portosystemic shunt tough re-circulation units. (2) in patients with membranous glomerulonephritis MGN serum complement levels were normal, no follow Ke: rheumatoid factor or performance cryoglobulinemia. chronic HCV infection may HCV protein domain heart caused by immune complex deposition in Taiwan. chronic HCV ① and skeletal muscle: joint pain is a common expression, some reports suggest that polymyositis and chronic HCV infection. i ② pulmonary fibrosis may be associated ③ Eye: HCV infection and Mooren corneal ulcers relationship yet to be confirmed. ④ salivary gland; Siogren syndrome often associated with ECM and HCV infection 3l · ⑤ skin include: vasculitis from the cell disruption (EMC ), lichen planus, the original fecal porphyrin psychosis. occasionally erythema nodosum, erythema multiforme, urticaria, Behcet syndrome, and white. @ cylinder tube: nodular hyperactivity Mi Yan. ⑦ Blood: Aplastic anemia in patients with acute viral hepatitis weeks to months,[link widoczny dla zalogowanych], often serious and irreversible. ⑤ nervous system: Guillain-Barre syndrome, multiple nerve root inflammation, cerebral infarctions, stupor, multiple single-neuritis,[link widoczny dla zalogowanych], peripheral neuropathy and cranial nerve paresis. ⑤ Thyroid: Hashimoto's thyroiditis and hypothyroidism @ lymphatic system B-cell non-Hodgkin's lymphoma may be a complication of chronic HCV infection. 3 stalk stomatitis virus influenza infection (1) simple red blood cells aplastic anemia, sub-acute severe hepatitis and easy platform wells pure red barrier, and high mortality. (2) thrombocytopenic purpura, optic neuritis, acute infectious polyneuritis, acute transverse myelitis, hemolytic anemia 4. Chronic hepatitis G virus infection and foot ths Zaidi and other reports 1 case, respectively, in patients with hepatitis G regeneration of bone marrow examination confirmed the Aplastic Anemia, Karayiam'fis found 4 sides HGV infected lymphocytes HGVRNA positive, indicating HGV is not only hepatotropic,[link widoczny dla zalogowanych], but also with cell tropism characteristics. extrahepatic manifestations of chronic hepatitis virus infection and impact of continuing mortality and mortality of HCV infected patients with chronic HBV infection in patients with chronic disorders to more common and associated with circulating autoantibodies Correction of immune disorders 3, 1999 publication on page 26, More articles related to topics：

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