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Palace compound net improvement of blood side effects of mifepristone Study

In 1XB2,[link widoczny dla zalogowanych], PGF1 levels and TXB2/PGFl value the role of trend,[link widoczny dla zalogowanych], in which the blood clear height Palace,[link widoczny dla zalogowanych], the dose could significantly reduce the drug flow in PGF1 levels in peripheral blood (P <0.05), Palace of blood dose was also significantly lower in height TXIh/PGF1 value (P <0.05); 相反, 0.05; P <0.01), Table 3. 2.4 Palace of net blood flow in rats on the drug decidual tissues and elastic fibers in rats and model group, the net house of the blood group of each dose of the drug flow to reduce residual rat decidua, and promote wound healing and increased endometrial sub-42 * Chengdu University of Traditional Chinese Medicine Volume 29, 2006 Palace of the role of trends in the number of elastic fibers in smooth muscle , which house the blood can significantly reduce the dose in height drug flow rat decidua residues. Net wrapped 3-rich blood to the large-l plasma TXB2, PCFl. Of (i ± s) shaping Tam Kui P <o. 053 discussion is generally believed that early pregnancy bleeding after medical abortion is not the medicine dripping The main pathogenesis is away from the uterus through the blood stasis, blood stasis do not, new blood ease, it is not just bleeding profusely; In addition, the stasis of a long and easy Bleeding profusely important factors is not absolute. Therefore, the treatment of this disease can not just consider the bleeding, but should blood circulation, heat and cooling blood to stop bleeding, based on in order to sample both. All parties from the blood of the net house angelica, Chuanxiong, peach kernel, Motherwort, purslane, Zhigancao composed of 6 herbs, on the basis of the biochemical soup to remove the rice wine, Tongbian, ginger and other drugs than the temperature of the dry, adding Motherwort and purslane enhance blood circulation, cooling blood to stop bleeding of power, can significantly reduce the clinical application of bleeding after mifepristone time, speed up the discharge of residual tissue. According to reports, after mifepristone, E2 level declines in uterine oxytocin sensitivity of the decline in uterine contraction poor decidual difficult to discharge the remaining bureaucratic L4J, led to delayed wound healing, endometrial, blood vessels can not be closed, the repeated exposure sinus membrane bleeding, pelvic congestion and stasis is always in the pathological state of bJ. Net increase in the level of blood E2 Palace one can promote endometrial wound healing, reduce bleeding directly; the other hand, can also increase uterine sensitivity to the oxytocin and strengthen contractions, and promote material discharge pregnancy, closed the bleeding point, indirectly, reduction of bleeding after medical abortion. Palace net increase P3H and LH blood levels, can inhibit the dissolution of mifepristone causes atrophy of the corpus luteum, and promote recovery of ovarian function, which indirectly promote the repair of the endometrium to reduce the drug flow m. P or lower than the constant lack of PRL expression may lead to early pregnancy miscarriage j, the official net decrease of blood P, PRL levels, contribute to abortion completely, reducing bleeding after medical abortion. The results showed that the net can promote drug Palace blood flow discharge of rat uterus and endometrial decidual tissue repair in rats significantly reduced the amount of bleeding. Palace of the mechanism of blood may be a net increase in uterine smooth muscle and its number of elastic fibers, increased E2, limp and LIt levels and lower P, PRL levels related. The results showed that net official blood flow in rats can reduce blood rIxB2/pGF1 drug value, indicating that the net may Palace blood running through the promotion and inhibition of blood platelet adhesion, aggregation and other functions, so that can not be firmly glued decidua in the uterus faster peeling off the membrane from the uterine cavity, and thus play the role of stasis to stop bleeding.
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